- ASSOCIATE PROFESSOR | Neurology
- ASSOCIATE PROFESSOR | Pathology
- ASSOCIATE PROFESSOR | Neurosurgery
- Hospital Affiliation
- The Mount Sinai Hospital
Dr. Zhou is a physician scientist. Her research team is actively exploring the molecular mechanisms underlying muscle inflammation and fibrosis associated with muscular dystrophy and acute skeletal muscle injury repair. Her research is aimed to develop novel pharmacotherapy to modify inflammation, reduce fibrosis, promote regeneration, and improve muscle function and phenotype in patients with muscular dystrophy. Her research is funded by the National Institute of Health.
- Amyotrophic Lateral Sclerosis
- Autonomic Disorder
- Brachial Plexopathy
- Carpal Tunnel Syndrome
- Cauda Equina Syndrome
- Charcot-Marie-Tooth Disease
- Chronic Inflammatory Demyelinating Polyneuropathy (CIDP)
- Chronic Neck Pain
- Complex Regional Pain Syndrome
- Cubital Tunnel Syndrome
- Diabetic Neuropathy
- Electrodiagnostic Testing (EMG/NCS)
- Foot Drop
- Gait Impairment
- Guillain-Barré Syndrome
- Herniated Disk
- Inclusion Body Myositis
- Inflammatory Myopathy
- Lambert-Eaton Myasthenic Syndrome
- Lumbar Radiculopathy
- Lumbosacral Plexopathy
- Lyme Disease
- Metabolic Myopathy
- Mitochondrial Myopathy
- Mononeuropathy Multiplex
- Motor Neuron Disease
- Multifocal Motor Neuropathy
- Muscular Dystrophy
- Myasthenia Gravis
- Neck Sprain
- Paraneoplastic Syndrome
- Periodic Paralysis Syndrome
- Peripheral Neuropathy
- Peroneal Nerve Injury
- Pinched Nerve
- Sensory Ganglionopathy
- Skin Biopsy
- Small Fiber Neuropathy
- Spinal Stenosis
- Toxic Myopathy
Fibrosis, Inflammation, Multiple Sclerosis, Muscular Dystrophy
Multi-Disciplinary Training Area
MD, Fudan University Shanghai Medical College
PhD, University of Cincinnati
Roles of chemokines and chemokine receptors in Duchenne muscular dystrophy
Duchenne muscular dystrophy (DMD) is the most common, lethal, and currently untreatable genetic muscle disease. Muscle pathology of DMD is featured by chronic inflammation and fibrosis, which directly lead to muscle dysfunction and clinical muscle weakness. Ameliorating muscle inflammation and fibrosis may thus represent a viable therapeutic approach for DMD. The chemokine system plays a key role in recruiting inflammatory cells and profibrogenic cells into injured muscles. This research is to determine the roles of the chemokine system in inflammation and fibrosis associated with DMD and to explore the possibility of modifying inflammation, reducing fibrosis, and improving muscle function by altering the expression and function of related chemokines and chemokine receptors. The DMD mouse models and relevant chemokine or chemokine receptor knockout mouse models are being used for the study.
Physicians and scientists on the faculty of the Icahn School of Medicine at Mount Sinai often interact with pharmaceutical, device and biotechnology companies to improve patient care, develop new therapies and achieve scientific breakthroughs. In order to promote an ethical and transparent environment for conducting research, providing clinical care and teaching, Mount Sinai requires that salaried faculty inform the School of their relationships with such companies.
Dr. Zhou has not yet completed reporting of Industry relationships.
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